Isocyanate-induced asthmatic reactions are associated with delayed increase in fractional exhaled nitric oxide measured at expiratory flow of 50mL/s (FeNO50), a biomarker of airway inflammation. The time-course of FeNO increase is compatible with activation of NO synthase, but the origin of NO production in the lung is undetermined.
Exhaled nitric oxide dynamics in asthmatic reactions induced by diisocyanates
MASON, PAOLAWriting – Original Draft Preparation
;SCARPA, MARIA CRISTINA;GUARNIERI, GABRIELLA;GIORDANO, GIUSEPPE;BARALDI, EUGENIO;MAESTRELLI, PIERO
2016
Abstract
Isocyanate-induced asthmatic reactions are associated with delayed increase in fractional exhaled nitric oxide measured at expiratory flow of 50mL/s (FeNO50), a biomarker of airway inflammation. The time-course of FeNO increase is compatible with activation of NO synthase, but the origin of NO production in the lung is undetermined.
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